DREAM Is a Critical Transcriptional Repressor for Pain Modulation

نویسندگان

  • Hai-Ying M. Cheng
  • Graham M. Pitcher
  • Steven R. Laviolette
  • Ian Q. Whishaw
  • Kit I. Tong
  • Lisa K. Kockeritz
  • Teiji Wada
  • Nicholas A. Joza
  • Michael Crackower
  • Jason Goncalves
  • Ildiko Sarosi
  • James R. Woodgett
  • Antonio J. Oliveira-dos-Santos
  • Mitsuhiko Ikura
  • Derek van der Kooy
  • Michael W. Salter
  • Josef M. Penninger
چکیده

Control and treatment of chronic pain remain major clinical challenges. Progress may be facilitated by a greater understanding of the mechanisms underlying pain processing. Here we show that the calcium-sensing protein DREAM is a transcriptional repressor involved in modulating pain. dream(-/-) mice displayed markedly reduced responses in models of acute thermal, mechanical, and visceral pain. dream(-/-) mice also exhibited reduced pain behaviors in models of chronic neuropathic and inflammatory pain. However, dream(-/-) mice showed no major defects in motor function or learning and memory. Mice lacking DREAM had elevated levels of prodynorphin mRNA and dynorphin A peptides in the spinal cord, and the reduction of pain behaviors in dream(-/-) mice was mediated through dynorphin-selective kappa (kappa)-opiate receptors. Thus, DREAM appears to be a critical transcriptional repressor in pain processing.

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عنوان ژورنال:
  • Cell

دوره 108  شماره 

صفحات  -

تاریخ انتشار 2002